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Glutathione works mainly in the liver, and it helps prevent cell damage and repair cells that have been damaged due to exposure to oxidative free radicals

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In a transgenic mouse model, depletion of FAP-expressing cells led to rapid hypoxic necrosis mediated by interferon-gamma (IFN-) and tumour necrosis factor-alpha (TNF-), both associated with CD8 + T cell-dependent tumour cell killing [47]

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